Cytokines and Chemokines in Infectious Diseases Handbook by Malak Kotb, Thierry Calandra

By Malak Kotb, Thierry Calandra

Univ. of Tennessee, Memphis. textual content presents a entire overview of cytokines and chemokines in immunity and human infectious illnesses. Covers bacterial, viral, fungal, and parasitic infections. comprises an review of healing innovations and brokers and the position in modulating host responses to pathogens and vaccines. DNLM: Chemokines--immunology.

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FINDINGS IN OTHER CYTOKINE CANDIDATE GENES Interleukin-10 shows well-defined haplotypic promoter variation. High IL-10 secretors presenting with fever of unknown origin display an increased mortality rate (48), whereas susceptibility to severe meningococcal disease as well as poor outcome seem to be linked to inherited high IL-10 secretion and low TNF release in a family study testing ex vivo cytokine inducibility (49). IL-10 promoter genotypes could not be associated with incidence or outcome of severe abdominal sepsis (Fig.

1998) Protection against lipoapoptosis of beta cells through leptin-dependent maintenance of Bcl-2 expression. Proc. Natl. Acad. Sci. USA 95, 9558–9561. 126. , et al. (1998) Plasma leptin levels are increased in survivors of acute sepsis: associated loss of diurnal rhythm, in cortisol and leptin secretion. J. Clin. Endocrinol. Metab. 83, 280–283. 127. , Madero, R. and Montiel, C. (1999) Relationship of plasma leptin to plasma cytokines and human survival in sepsis and septic shock. J. Infect. Dis.

119. , Talabot-Ayer, D. et al. (2002) Leptin signaling deficiency impairs humoral and cellular immune responses and attenuates experimental arthritis. J. Immunol. 168, 875–882. 120. , et al. (2001) Leptin potentiates experimental autoimmune encephalomyelitis in SJL female mice and confers susceptibility to males. Eur. J. Immunol. 31, 1324–1332. 121. , et al. (2002) Balancing susceptibility to infection and autoimmunity: a role for leptin? Trends Immunol. 23, 182–187. 122. I. (2002) Leptin inhibits the anti-CD3 driven proliferation of peripheral blood T cells but enhances the production of pro-inflammatory cytokines.

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